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In recent years, research has demonstrated that activation of GLP-1R produces multifaceted cardiovascular protective effects independent of its hypoglycemic action, specifically including improved vascular endothelial function, inhibition of atherosclerotic plaque formation, modulation of the body’s inflammatory response, alleviation of oxidative stress damage, and improved myocardial energy metabolism (8, 9)

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Laboratory studies use 5mg, 10mg, and 15mg doses to investigate dose-dependent effects, with the peptide’s stability allowing precise observation of slow signaling pathways